Science Magazine published the September 2026 issue of Science Immunology featuring a cover depicting neutrophils dynamically altering their actomyosin properties to migrate quickly through narrow artificial capillaries.
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The September 2026 issue of #ScienceImmunology is out! This month's cover depicts neutrophils dynamically altering their actomyosin properties to migrate quickly through…
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Science Magazine
Cutting-edge research, news, commentary, and visuals from the Science family of journals.
- Industry
- Book and Periodical Publishing
- Location
- Washington, US
- Company size
- 201–500 employees
About Science Magazine
Science has been at the center of important scientific discovery since its founding in 1880. Today, Science continues to publish the very best in research across the sciences, with articles that consistently rank among the most cited in the world. A trailblazer in online publishing as well, the Science family of publications include online journals Science Translational Medicine, Science Signaling, Science Immunology, Science Robotics, and the open access journal Science Advances. The Science family of journals is published by the American Association for the Advancement of Science (AAAS), the world’s oldest and largest general science organization. The nonprofit AAAS serves 10 million people through primary memberships and affiliations with some 262 scientific societies and academies.
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Presence & Recognition
Science Magazine reported that the Ig Nobel Prize winners accepted their prizes in Zürich for the first time in the Ig Nobel's 36-year history, not in the Boston area.
Presence & Recognition
Science Magazine featured a segment on the Science Magazine Podcast where Sarah Crespi spoke to Veronika Bókony about temperature-dependent sex determination in amphibians and their implications in a warming world.
Research & Knowledge
Science Magazine published a study in Science showing that chemically enhanced suppressor tRNAs combined with a lung-targeted delivery system can restore production of a critical protein in models of cystic fibrosis caused by nonsense mutations.
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